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Most of the studies reviewed used doses that were twice the recommended amount -- 50 milligrams -- of diphenhydramine, the active ingredient in Benadryl and other first-generation "sedating" allergy medications.
"We're not saying that there's not a difference," says lead researcher Bruce G.
Because the new class of non-sedating antihistamines is far more accurate than its predecessors in terms of targeting specific nerve cell receptors, there is a much lower incidence of non-sedating antihistamines binding to inappropriate cells, such as certain ones in the brain that are responsible for causing sedation when activated.
Non-sedating antihistamines actually have the capacity to avoid them.
It is the release of histamine that causes the body to protect itself from the pathogens in ways that we recognize as an allergic reaction.
Histamine is responsible for the immune response to the stimuli that we are familiar in symptoms as itching, sneezing, hives, runny nose and watery eyes. Traditional antihistamines act by minimizing swelling and controlling the dilation of small blood vessels that translate to those common allergy symptoms.
Non-sedating antihistamines are as effective as others but offer additional benefits.
The most significant advantage of non-sedating antihistamines is just what their mane implies: they do not have a sedative-like effect on the user.
In effect, this prevents them from acting as they would normally and releasing the histamine.They accomplished this by blocking the histamine from connecting with its receptors in specific nerve, muscle and glandular types of cells.There are some others as well that play a less significant role.contains one of the largest antihistamine dedicated article libraries, containing information ranging from allergies to antihistamine overdose issues.To get started, click on any of the links below to get started.